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We propose a dynamical model in which a network structure evolves in a self-organized critical (SOC) manner and explain a possible origin of the emergence of fractal and small-world networks. Our model combines a network growth and its decay by failures of nodes. The decay mechanism reflects the instability of large functional networks against cascading overload failures. It is demonstrated that the dynamical system surely exhibits SOC characteristics, such as power-law forms of the avalanche size distribution, the cluster size distribution, and the distribution of the time interval between intermittent avalanches. During the network evolution, fractal networks are spontaneously generated when networks experience critical cascades of failures that lead to a percolation transition. In contrast, networks far from criticality have small-world structures. We also observe the crossover behavior from fractal to small-world structure in the network evolution.
In this paper, a simple dynamical model in which fractal networks are formed by self-organized critical (SOC) dynamics is proposed; the proposed model consists of growth and collapse processes. It has been shown that SOC dynamics are realized by the
Self-organized bistability (SOB) is the counterpart of self-organized criticality (SOC), for systems tuning themselves to the edge of bistability of a discontinuous phase transition, rather than to the critical point of a continuous one. The equation
We explore simple models aimed at the study of social contagion, in which contagion proceeds through two stages. When coupled with demographic turnover, we show that two-stage contagion leads to nonlinear phenomena which are not present in the basic
Productive societies feature high levels of cooperation and strong connections between individuals. Public Goods Games (PGGs) are frequently used to study the development of social connections and cooperative behavior in model societies. In such game
We introduce a mathematical description of the impact of sociality in the spread of infectious diseases by integrating an epidemiological dynamics with a kinetic modeling of population-based contacts. The kinetic description leads to study the evolut