ترغب بنشر مسار تعليمي؟ اضغط هنا

The Causal-Neural Connection: Expressiveness, Learnability, and Inference

359   0   0.0 ( 0 )
 نشر من قبل Kevin Xia
 تاريخ النشر 2021
  مجال البحث الهندسة المعلوماتية
والبحث باللغة English




اسأل ChatGPT حول البحث

One of the central elements of any causal inference is an object called structural causal model (SCM), which represents a collection of mechanisms and exogenous sources of random variation of the system under investigation (Pearl, 2000). An important property of many kinds of neural networks is universal approximability: the ability to approximate any function to arbitrary precision. Given this property, one may be tempted to surmise that a collection of neural nets is capable of learning any SCM by training on data generated by that SCM. In this paper, we show this is not the case by disentangling the notions of expressivity and learnability. Specifically, we show that the causal hierarchy theorem (Thm. 1, Bareinboim et al., 2020), which describes the limits of what can be learned from data, still holds for neural models. For instance, an arbitrarily complex and expressive neural net is unable to predict the effects of interventions given observational data alone. Given this result, we introduce a special type of SCM called a neural causal model (NCM), and formalize a new type of inductive bias to encode structural constraints necessary for performing causal inferences. Building on this new class of models, we focus on solving two canonical tasks found in the literature known as causal identification and estimation. Leveraging the neural toolbox, we develop an algorithm that is both sufficient and necessary to determine whether a causal effect can be learned from data (i.e., causal identifiability); it then estimates the effect whenever identifiability holds (causal estimation). Simulations corroborate the proposed approach.



قيم البحث

اقرأ أيضاً

Several recent works have shown separation results between deep neural networks, and hypothesis classes with inferior approximation capacity such as shallow networks or kernel classes. On the other hand, the fact that deep networks can efficiently ex press a target function does not mean that this target function can be learned efficiently by deep neural networks. In this work we study the intricate connection between learnability and approximation capacity. We show that learnability with deep networks of a target function depends on the ability of simpler classes to approximate the target. Specifically, we show that a necessary condition for a function to be learnable by gradient descent on deep neural networks is to be able to approximate the function, at least in a weak sense, with shallow neural networks. We also show that a class of functions can be learned by an efficient statistical query algorithm if and only if it can be approximated in a weak sense by some kernel class. We give several examples of functions which demonstrate depth separation, and conclude that they cannot be efficiently learned, even by a hypothesis class that can efficiently approximate them.
Constraint-based causal discovery from limited data is a notoriously difficult challenge due to the many borderline independence test decisions. Several approaches to improve the reliability of the predictions by exploiting redundancy in the independ ence information have been proposed recently. Though promising, existing approaches can still be greatly improved in terms of accuracy and scalability. We present a novel method that reduces the combinatorial explosion of the search space by using a more coarse-grained representation of causal information, drastically reducing computation time. Additionally, we propose a method to score causal predictions based on their confidence. Crucially, our implementation also allows one to easily combine observational and interventional data and to incorporate various types of available background knowledge. We prove soundness and asymptotic consistency of our method and demonstrate that it can outperform the state-of-the-art on synthetic data, achieving a speedup of several orders of magnitude. We illustrate its practical feasibility by applying it on a challenging protein data set.
Classical causal and statistical inference methods typically assume the observed data consists of independent realizations. However, in many applications this assumption is inappropriate due to a network of dependences between units in the data. Meth ods for estimating causal effects have been developed in the setting where the structure of dependence between units is known exactly, but in practice there is often substantial uncertainty about the precise network structure. This is true, for example, in trial data drawn from vulnerable communities where social ties are difficult to query directly. In this paper we combine techniques from the structure learning and interference literatures in causal inference, proposing a general method for estimating causal effects under data dependence when the structure of this dependence is not known a priori. We demonstrate the utility of our method on synthetic datasets which exhibit network dependence.
Data scarcity is a tremendous challenge in causal effect estimation. In this paper, we propose to exploit additional data sources to facilitate estimating causal effects in the target population. Specifically, we leverage additional source datasets w hich share similar causal mechanisms with the target observations to help infer causal effects of the target population. We propose three levels of knowledge transfer, through modelling the outcomes, treatments, and confounders. To achieve consistent positive transfer, we introduce learnable parametric transfer factors to adaptively control the transfer strength, and thus achieving a fair and balanced knowledge transfer between the sources and the target. The proposed method can infer causal effects in the target population without prior knowledge of data discrepancy between the additional data sources and the target. Experiments on both synthetic and real-world datasets show the effectiveness of the proposed method as compared with recent baselines.
Learning the causal structure that underlies data is a crucial step towards robust real-world decision making. The majority of existing work in causal inference focuses on determining a single directed acyclic graph (DAG) or a Markov equivalence clas s thereof. However, a crucial aspect to acting intelligently upon the knowledge about causal structure which has been inferred from finite data demands reasoning about its uncertainty. For instance, planning interventions to find out more about the causal mechanisms that govern our data requires quantifying epistemic uncertainty over DAGs. While Bayesian causal inference allows to do so, the posterior over DAGs becomes intractable even for a small number of variables. Aiming to overcome this issue, we propose a form of variational inference over the graphs of Structural Causal Models (SCMs). To this end, we introduce a parametric variational family modelled by an autoregressive distribution over the space of discrete DAGs. Its number of parameters does not grow exponentially with the number of variables and can be tractably learned by maximising an Evidence Lower Bound (ELBO). In our experiments, we demonstrate that the proposed variational posterior is able to provide a good approximation of the true posterior.

الأسئلة المقترحة

التعليقات
جاري جلب التعليقات جاري جلب التعليقات
سجل دخول لتتمكن من متابعة معايير البحث التي قمت باختيارها
mircosoft-partner

هل ترغب بارسال اشعارات عن اخر التحديثات في شمرا-اكاديميا