ترغب بنشر مسار تعليمي؟ اضغط هنا

Origins of power-law degree distribution in the heterogeneity of human activity in social networks

128   0   0.0 ( 0 )
 نشر من قبل Sen Pei
 تاريخ النشر 2013
  مجال البحث فيزياء
والبحث باللغة English




اسأل ChatGPT حول البحث

The probability distribution of number of ties of an individual in a social network follows a scale-free power-law. However, how this distribution arises has not been conclusively demonstrated in direct analyses of peoples actions in social networks. Here, we perform a causal inference analysis and find an underlying cause for this phenomenon. Our analysis indicates that heavy-tailed degree distribution is causally determined by similarly skewed distribution of human activity. Specifically, the degree of an individual is entirely random - following a maximum entropy attachment model - except for its mean value which depends deterministically on the volume of the users activity. This relation cannot be explained by interactive models, like preferential attachment, since the observed actions are not likely to be caused by interactions with other people.



قيم البحث

اقرأ أيضاً

135 - J. Xie , J. Emenheiser , M. Kirby 2011
Public opinion is often affected by the presence of committed groups of individuals dedicated to competing points of view. Using a model of pairwise social influence, we study how the presence of such groups within social networks affects the outcome and the speed of evolution of the overall opinion on the network. Earlier work indicated that a single committed group within a dense social network can cause the entire network to quickly adopt the groups opinion (in times scaling logarithmically with the network size), so long as the committed group constitutes more than about 10% of the population (with the findings being qualitatively similar for sparse networks as well). Here we study the more general case of opinion evolution when two groups committed to distinct, competing opinions $A$ and $B$, and constituting fractions $p_A$ and $p_B$ of the total population respectively, are present in the network. We show for stylized social networks (including ErdH{o}s-Renyi random graphs and Barabasi-Albert scale-free networks) that the phase diagram of this system in parameter space $(p_A,p_B)$ consists of two regions, one where two stable steady-states coexist, and the remaining where only a single stable steady-state exists. These two regions are separated by two fold-bifurcation (spinodal) lines which meet tangentially and terminate at a cusp (critical point). We provide further insights to the phase diagram and to the nature of the underlying phase transitions by investigating the model on infinite (mean-field limit), finite complete graphs and finite sparse networks. For the latter case, we also derive the scaling exponent associated with the exponential growth of switching times as a function of the distance from the critical point.
We develop a theoretical framework for the study of epidemic-like social contagion in large scale social systems. We consider the most general setting in which different communication platforms or categories form multiplex networks. Specifically, we propose a contact-based information spreading model, and show that the critical point of the multiplex system associated to the active phase is determined by the layer whose contact probability matrix has the largest eigenvalue. The framework is applied to a number of different situations, including a real multiplex system. Finally, we also show that when the system through which information is disseminating is inherently multiplex, working with the graph that results from the aggregation of the different layers is flawed.
We study the effect of heterogeneous temporal activations on epidemic spreading in temporal networks. We focus on the susceptible-infected-susceptible (SIS) model on activity-driven networks with burstiness. By using an activity-based mean-field appr oach, we derive a closed analytical form for the epidemic threshold for arbitrary activity and inter-event time distributions. We show that, as expected, burstiness lowers the epidemic threshold while its effect on prevalence is twofold. In low-infective systems burstiness raises the average infection probability, while it weakens epidemic spreading for high infectivity. Our results can help clarify the conflicting effects of burstiness reported in the literature. We also discuss the scaling properties at the transition, showing that they are not affected by burstiness.
We study the robustness properties of multiplex networks consisting of multiple layers of distinct types of links, focusing on the role of correlations between degrees of a node in different layers. We use generating function formalism to address var ious notions of the network robustness relevant to multiplex networks such as the resilience of ordinary- and mutual connectivity under random or targeted node removals as well as the biconnectivity. We found that correlated coupling can affect the structural robustness of multiplex networks in diverse fashion. For example, for maximally-correlated duplex networks, all pairs of nodes in the giant component are connected via at least two independent paths and network structure is highly resilient to random failure. In contrast, anti-correlated duplex networks are on one hand robust against targeted attack on high-degree nodes, but on the other hand they can be vulnerable to random failure.
We propose a bare-bones stochastic model that takes into account both the geographical distribution of people within a country and their complex network of connections. The model, which is designed to give rise to a scale-free network of social conne ctions and to visually resemble the geographical spread seen in satellite pictures of the Earth at night, gives rise to a power-law distribution for the ranking of cities by population size (but for the largest cities) and reflects the notion that highly connected individuals tend to live in highly populated areas. It also yields some interesting insights regarding Gibrats law for the rates of city growth (by population size), in partial support of the findings in a recent analysis of real data [Rozenfeld et al., Proc. Natl. Acad. Sci. U.S.A. 105, 18702 (2008)]. The model produces a nontrivial relation between city population and city population density and a superlinear relationship between social connectivity and city population, both of which seem quite in line with real data.
التعليقات
جاري جلب التعليقات جاري جلب التعليقات
سجل دخول لتتمكن من متابعة معايير البحث التي قمت باختيارها
mircosoft-partner

هل ترغب بارسال اشعارات عن اخر التحديثات في شمرا-اكاديميا