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Register a new userMeasurement-Induced Boolean Dynamics and Controllability for Quantum Networks
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In this paper, we study dynamical quantum networks which evolve according to Schrodinger equations but subject to sequential local or global quantum measurements. A network of qubits forms a composite quantum system whose state undergoes unitary evolution in between periodic measurements, leading to hybrid quantum dynamics with random jumps at discrete time instances along a continuous orbit. The measurements either act on the entire network of qubits, or only a subset of qubits. First of all, we reveal that this type of hybrid quantum dynamics induces probabilistic Boolean recursions representing the measurement outcomes. With global measurements, it is shown that such resulting Boolean recursions define Markov chains whose state-transitions are fully determined by the network Hamiltonian and the measurement observables. Particularly, we establish an explicit and algebraic representation of the underlying recursive random mapping driving such induced Markov chains. Next, with local measurements, the resulting probabilistic Boolean dynamics is shown to be no longer Markovian. The state transition probability at any given time becomes dependent on the entire history of the sample path, for which we establish a recursive way of computing such non-Markovian probability transitions. Finally, we adopt the classical bilinear control model for the continuous Schrodinger evolution, and show how the measurements affect the controllability of the quantum networks.
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In this paper, we study the recursion of measurement outcomes for open quantum networks under sequential measurements. Open quantum networks are networked quantum subsystems (e.g., qubits) with the state evolutions described by a continuous Lindblad master equation. When measurements are performed sequentially along such continuous dynamics, the quantum network states undergo random jumps and the corresponding measurement outcomes can be described by a vector of probabilistic Boolean variables. The induced recursion of the Boolean vectors forms a probabilistic Boolean network. First of all, we show that the state transition of the induced Boolean networks can be explicitly represented through realification of the master equation. Next, when the open quantum dynamics is relaxing in the sense that it possesses a unique equilibrium as a global attractor, structural properties including absorbing states, reducibility, and periodicity for the induced Boolean network are direct consequences of the relaxing property. Particularly, we show that generically, relaxing quantum dynamics leads to irreducible and aperiodic chains for the measurement outcomes. Finally, we show that for quantum consensus networks as a type of non-relaxing open quantum network dynamics, the communication classes of the measurement-induced Boolean networks are encoded in the quantum Laplacian of the underlying interaction graph.
Cancer forms a robust system and progresses as stages over time typically with increasing aggressiveness and worsening prognosis. Characterizing these stages and identifying the genes driving transitions between them is critical to understand cancer progression and to develop effective anti-cancer therapies. Here, we propose a novel model of the cancer system as a Boolean state space in which a Boolean network, built from protein interaction and gene-expression data from different stages of cancer, transits between Boolean satisfiability states by editing interactions and flipping genes. The application of our model (called BoolSpace) on three case studies - pancreatic and breast tumours in human and post spinal-cord injury in rats - reveals valuable insights into the phenomenon of cancer progression. In particular, we notice that several of the genes flipped are serine/threonine kinases which act as natural cellular switches and that different sets of genes are flipped during the initial and final stages indicating a pattern to tumour progression. We hypothesize that robustness of cancer partly stems from passing of the baton between genes at different stages, and therefore an effective therapy should target a cover set of these genes. A C/C++ implementation of BoolSpace is freely available at: http://www.bioinformatics.org.au/tools-data
Observabililty is an important topic of Boolean control networks (BCNs). In this paper, we propose a new type of observability named online observability to present the sufficient and necessary condition of determining the initial states of BCNs, when their initial states cannot be reset. And we design an algorithm to decide whether a BCN has the online observability. Moreover, we prove that a BCN is identifiable iff it satisfies controllability and the online observability, which reveals the essence of identification problem of BCNs.
In this paper we consider the problem of controlling a limited number of target nodes of a network. Equivalently, we can see this problem as controlling the target variables of a structured system, where the state variables of the system are associated to the nodes of the network. We deal with this problem from a different point of view as compared to most recent literature. Indeed, instead of considering controllability in the Kalman sense, that is, as the ability to drive the target states to a desired value, we consider the stronger requirement of driving the target variables as time functions. The latter notion is called functional target controllability. We think that restricting the controllability requirement to a limited set of important variables justifies using a more accurate notion of controllability for these variables. Remarkably, the notion of functional controllability allows formulating very simple graphical conditions for target controllability in the spirit of the structural approach to controllability. The functional approach enables us, moreover, to determine the smallest set of steering nodes that need to be actuated to ensure target controllability, where these steering nodes are constrained to belong to a given set. We show that such a smallest set can be found in polynomial time. We are also able to classify the possible actuated variables in terms of their importance with respect to the functional target controllability problem.
We consider a dynamic protocol for quantum many-body systems, which enables to study the interplay between unitary Hamiltonian driving and random local projective measurements. While the unitary dynamics tends to increase entanglement, local measurements tend to disentangle, thus favoring decoherence. Close to a quantum transition where the system develops critical correlations with diverging length scales, the competition of the two drivings is analyzed within a dynamic scaling framework, allowing us to identify a regime (dynamic scaling limit) where the two mechanisms develop a nontrivial interplay. We perform a numerical analysis of this protocol in a measurement-driven Ising chain, which supports the scaling laws we put forward. The local measurement process generally tends to suppress quantum correlations, even in the dynamic scaling limit. The power law of the decay of the quantum correlations turns out to be enhanced at the quantum transition.
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