No Arabic abstract
There has been a rich interplay in recent years between (i) empirical investigations of real world dynamic networks, (ii) analytical modeling of the microscopic mechanisms that drive the emergence of such networks, and (iii) harnessing of these mechanisms to either manipulate existing networks, or engineer new networks for specific tasks. We continue in this vein, and study the deletion phenomenon in the web by following two different sets of web-sites (each comprising more than 150,000 pages) over a one-year period. Empirical data show that there is a significant deletion component in the underlying web networks, but the deletion process is not uniform. This motivates us to introduce a new mechanism of preferential survival (PS), where nodes are removed according to a degree-dependent deletion kernel. We use the mean-field rate equation approach to study a general dynamic model driven by Preferential Attachment (PA), Double PA (DPA), and a tunable PS, where c nodes (c<1) are deleted per node added to the network, and verify our predictions via large-scale simulations. One of our results shows that, unlike in the case of uniform deletion, the PS kernel when coupled with the standard PA mechanism, can lead to heavy-tailed power law networks even in the presence of extreme turnover in the network. Moreover, a weak DPA mechanism, coupled with PS, can help make the network even more heavy-tailed, especially in the limit when deletion and insertion rates are almost equal, and the overall network growth is minimal. The dynamics reported in this work can be used to design and engineer stable ad hoc networks and explain the stability of the power law exponents observed in real-world networks.
In this paper we introduce a family of planar, modular and self-similar graphs which have small-world and scale-free properties. The main parameters of this family are comparable to those of networks associated to complex systems, and therefore the graphs are of interest as mathematical models for these systems. As the clustering coefficient of the graphs is zero, this family is an explicit construction that does not match the usual characterization of hierarchical modular networks, namely that vertices have clustering values inversely proportional to their degrees.
Complex networks have acquired a great popularity in recent years, since the graph representation of many natural, social and technological systems is often very helpful to characterize and model their phenomenology. Additionally, the mathematical tools of statistical physics have proven to be particularly suitable for studying and understanding complex networks. Nevertheless, an important obstacle to this theoretical approach is still represented by the difficulties to draw parallelisms between network science and more traditional aspects of statistical physics. In this paper, we explore the relation between complex networks and a well known topic of statistical physics: renormalization. A general method to analyze renormalization flows of complex networks is introduced. The method can be applied to study any suitable renormalization transformation. Finite-size scaling can be performed on computer-generated networks in order to classify them in universality classes. We also present applications of the method on real networks.
In recent years the research community has accumulated overwhelming evidence for the emergence of complex and heterogeneous connectivity patterns in a wide range of biological and sociotechnical systems. The complex properties of real-world networks have a profound impact on the behavior of equilibrium and nonequilibrium phenomena occurring in various systems, and the study of epidemic spreading is central to our understanding of the unfolding of dynamical processes in complex networks. The theoretical analysis of epidemic spreading in heterogeneous networks requires the development of novel analytical frameworks, and it has produced results of conceptual and practical relevance. A coherent and comprehensive review of the vast research activity concerning epidemic processes is presented, detailing the successful theoretical approaches as well as making their limits and assumptions clear. Physicists, mathematicians, epidemiologists, computer, and social scientists share a common interest in studying epidemic spreading and rely on similar models for the description of the diffusion of pathogens, knowledge, and innovation. For this reason, while focusing on the main results and the paradigmatic models in infectious disease modeling, the major results concerning generalized social contagion processes are also presented. Finally, the research activity at the forefront in the study of epidemic spreading in coevolving, coupled, and time-varying networks is reported.
The availability of data from many different sources and fields of science has made it possible to map out an increasing number of networks of contacts and interactions. However, quantifying how reliable these data are remains an open problem. From Biology to Sociology and Economy, the identification of false and missing positives has become a problem that calls for a solution. In this work we extend one of newest, best performing models -due to Guimera and Sales-Pardo in 2009- to directed networks. The new methodology is able to identify missing and spurious directed interactions, which renders it particularly useful to analyze data reliability in systems like trophic webs, gene regulatory networks, communication patterns and social systems. We also show, using real-world networks, how the method can be employed to help searching for new interactions in an efficient way.
We introduce a general stochastic model for the spread of rumours, and derive mean-field equations that describe the dynamics of the model on complex social networks (in particular those mediated by the Internet). We use analytical and numerical solutions of these equations to examine the threshold behavior and dynamics of the model on several models of such networks: random graphs, uncorrelated scale-free networks and scale-free networks with assortative degree correlations. We show that in both homogeneous networks and random graphs the model exhibits a critical threshold in the rumour spreading rate below which a rumour cannot propagate in the system. In the case of scale-free networks, on the other hand, this threshold becomes vanishingly small in the limit of infinite system size. We find that the initial rate at which a rumour spreads is much higher in scale-free networks than in random graphs, and that the rate at which the spreading proceeds on scale-free networks is further increased when assortative degree correlations are introduced. The impact of degree correlations on the final fraction of nodes that ever hears a rumour, however, depends on the interplay between network topology and the rumour spreading rate. Our results show that scale-free social networks are prone to the spreading of rumours, just as they are to the spreading of infections. They are relevant to the spreading dynamics of chain emails, viral advertising and large-scale information dissemination algorithms on the Internet.