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The recent discovery of Jupiter-mass planets orbiting at a few AU from their stars compliments earlier detections of massive planets on very small orbits. The short period orbits strongly suggest that planet migration has occurred, with the likely mechanism being tidal interactions between the planets and the gas disks out of which they formed. The newly discovered long period planets, together with the gas giant planets in our solar system, show that migration is either absent or rapidly halted in at least some systems. We propose a mechanism for halting type-II migration at several AU in a gas disk. Photoevaporation of the disk by irradiation from the central star can produce a gap in the disk at a few AU, preventing planets outside the gap from migrating down to the star. This would result in an excess of systems with planets at or just outside the photoevaporation radius.
The discovery of giant planets in wide orbits represents a major challenge for planet formation theory. In the standard core accretion paradigm planets are expected to form at radial distances $lesssim 20$ au in order to form massive cores (with mass
We calculate the rate of photoevaporation of a circumstellar disk by energetic radiation (FUV, 6eV $<h u<$13.6eV; EUV, 13.6eV $<h u<$0.1keV; and Xrays, $h u>0.1$keV) from its central star. We focus on the effects of FUV and X-ray photons since EUV ph
The mass-period or radius-period distribution of close-in exoplanets shows a paucity of intermediate mass/size (sub-Jovian) planets with periods ~< 3 days. We show that this sub-Jovian desert can be explained by the photoevaporation of highly irradia
We present the time evolution of viscously accreting circumstellar disks as they are irradiated by ultraviolet and X-ray photons from a low-mass central star. Our model is a hybrid of a 1D time-dependent viscous disk model coupled to a 1+1D disk vert
Transition discs are expected to be a natural outcome of the interplay between photoevaporation (PE) and giant planet formation. Massive planets reduce the inflow of material from the outer to the inner disc, therefore triggering an earlier onset of