ترغب بنشر مسار تعليمي؟ اضغط هنا

Synchronization in time-varying networks

115   0   0.0 ( 0 )
 نشر من قبل Karin Alfaro-Bittner
 تاريخ النشر 2021
  مجال البحث فيزياء
والبحث باللغة English




اسأل ChatGPT حول البحث

Over the past two decades, complex network theory provided the ideal framework for investigating the intimate relationships between the topological properties characterizing the wiring of connections among a systems unitary components and its emergent synchronized functioning. An increased number of setups from the real world found therefore a representation in term of graphs, while more and more sophisticated methods were developed with the aim of furnishing a realistic description of the connectivity patterns under study. In particular, a significant number of systems in physics, biology and social science features a time-varying nature of the interactions among their units. We here give a comprehensive review of the major results obtained by contemporary studies on the emergence of synchronization in time-varying networks. In particular, two paradigmatic frameworks will be described in details. The first encompasses those systems where the time dependence of the nodes connections is due to adaptation, external forces, or any other process affecting each of the links of the network. The second framework, instead, corresponds to the case in which the structural evolution of the graph is due to the movement of the nodes, or agents, in physical spaces and to the fact that interactions may be ruled by space-dependent laws in a way that connections are continuously switched on and off in the course of the time. Finally, our report ends with a short discussion on promising directions and open problems for future studies.

قيم البحث

اقرأ أيضاً

The metapopulation framework is adopted in a wide array of disciplines to describe systems of well separated yet connected subpopulations. The subgroups or patches are often represented as nodes in a network whose links represent the migration routes among them. The connections have been so far mostly considered as static, but in general evolve in time. Here we address this case by investigating simple contagion processes on time-varying metapopulation networks. We focus on the SIR process and determine analytically the mobility threshold for the onset of an epidemic spreading in the framework of activity-driven network models. We find profound differences from the case of static networks. The threshold is entirely described by the dynamical parameters defining the average number of instantaneously migrating individuals and does not depend on the properties of the static network representation. Remarkably, the diffusion and contagion processes are slower in time-varying graphs than in their aggregated static counterparts, the mobility threshold being even two orders of magnitude larger in the first case. The presented results confirm the importance of considering the time-varying nature of complex networks.
The vast majority of strategies aimed at controlling contagion processes on networks considers the connectivity pattern of the system as either quenched or annealed. However, in the real world many networks are highly dynamical and evolve in time con currently to the contagion process. Here, we derive an analytical framework for the study of control strategies specifically devised for time-varying networks. We consider the removal/immunization of individual nodes according the their activity in the network and develop a block variable mean-field approach that allows the derivation of the equations describing the evolution of the contagion process concurrently to the network dynamic. We derive the critical immunization threshold and assess the effectiveness of the control strategies. Finally, we validate the theoretical picture by simulating numerically the information spreading process and control strategies in both synthetic networks and a large-scale, real-world mobile telephone call dataset
We investigate the effects of modular and temporal connectivity patterns on epidemic spreading. To this end, we introduce and analytically characterise a model of time-varying networks with tunable modularity. Within this framework, we study the epid emic size of Susceptible-Infected-Recovered, SIR, models and the epidemic threshold of Susceptible-Infected-Susceptible, SIS, models. Interestingly, we find that while the presence of tightly connected clusters inhibit SIR processes, it speeds up SIS diseases. In this case, we observe that heterogeneous temporal connectivity patterns and modular structures induce a reduction of the threshold with respect to time-varying networks without communities. We confirm the theoretical results by means of extensive numerical simulations both on synthetic graphs as well as on a real modular and temporal network.
78 - Hyewon Kim , Meesoon Ha , 2015
The formation of network structure is mainly influenced by an individual nodes activity and its memory, where activity can usually be interpreted as the individual inherent property and memory can be represented by the interaction strength between no des. In our study, we define the activity through the appearance pattern in the time-aggregated network representation, and quantify the memory through the contact pattern of empirical temporal networks. To address the role of activity and memory in epidemics on time-varying networks, we propose temporal-pattern coarsening of activity-driven growing networks with memory. In particular, we focus on the relation between time-scale coarsening and spreading dynamics in the context of dynamic scaling and finite-size scaling. Finally, we discuss the universality issue of spreading dynamics on time-varying networks for various memory-causality tests.
Social interactions are stratified in multiple contexts and are subject to complex temporal dynamics. The systematic study of these two features of social systems has started only very recently mainly thanks to the development of multiplex and time-v arying networks. However, these two advancements have progressed almost in parallel with very little overlap. Thus, the interplay between multiplexity and the temporal nature of connectivity patterns is poorly understood. Here, we aim to tackle this limitation by introducing a time-varying model of multiplex networks. We are interested in characterizing how these two properties affect contagion processes. To this end, we study SIS epidemic models unfolding at comparable time-scale respect to the evolution of the multiplex network. We study both analytically and numerically the epidemic threshold as a function of the overlap between, and the features of, each layer. We found that, the overlap between layers significantly reduces the epidemic threshold especially when the temporal activation patterns of overlapping nodes are positively correlated. Furthermore, when the average connectivity across layers is very different, the contagion dynamics are driven by the features of the more densely connected layer. Here, the epidemic threshold is equivalent to that of a single layered graph and the impact of the disease, in the layer driving the contagion, is independent of the overlap. However, this is not the case in the other layers where the spreading dynamics are sharply influenced by it. The results presented provide another step towards the characterization of the properties of real networks and their effects on contagion phenomena
التعليقات
جاري جلب التعليقات جاري جلب التعليقات
سجل دخول لتتمكن من متابعة معايير البحث التي قمت باختيارها
mircosoft-partner

هل ترغب بارسال اشعارات عن اخر التحديثات في شمرا-اكاديميا