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In this paper, we study the influence of the mortality trade-off in a nonlocal reaction-diffusion-mutation equation that we introduce to model the invasion of cane toads in Australia. This model is built off of one that has attracted attention recently, in which the population of toads is structured by a phenotypical trait that governs the spatial diffusion. We are concerned with the case when the diffusivity can take unbounded values and the mortality trade-off depends only on the trait variable. Depending on the rate of increase of the penalization term, we obtain the rate of spreading of the population. We identify two regimes, an acceleration regime when the penalization is weak and a linear spreading regime when the penalization is strong. While the development of the model comes from biological principles, the bulk of the article is dedicated to the mathematical analysis of the model, which is very technical.
We study the asymptotic spreading of Kolmogorov-Petrovsky-Piskunov (KPP) fronts in heterogeneous shifting habitats, with any number of shifting speeds, by further developing the method based on the theory of viscosity solutions of Hamilton-Jacobi equ
We establish spreading properties of the Lotka-Volterra competition-diffusion system. When the initial data vanish on a right half-line, we derive the exact spreading speeds and prove the convergence to homogeneous equilibrium states between successi
This is part two of our study on the spreading properties of the Lotka-Volterra competition-diffusion systems with a stable coexistence state. We focus on the case when the initial data are exponential decaying. By establishing a comparison principle
With great theoretical and practical significance, identifying the node spreading influence of complex network is one of the most promising domains. So far, various topology-based centrality measures have been proposed to identify the node spreading
The spreading of bacterial populations is central to processes in agriculture, the environment, and medicine. However, existing models of spreading typically focus on cells in unconfined settings--despite the fact that many bacteria inhabit complex a