No Arabic abstract
Motivated by the importance of individual differences in risk perception and behavior change in peoples responses to infectious disease outbreaks (particularly the ongoing COVID-19 pandemic), we propose a heterogeneous Disease-Behavior-Information (hDBI) transmission model, in which peoples risk of getting infected is influenced by information diffusion, behavior change, and disease transmission. We use both a mean-field approximation and Monte Carlo simulations to analyze the dynamics of the model. Information diffusion influences behavior change by allowing people to be aware of the disease and adopt self-protection, and subsequently affects disease transmission by changing the actual infection rate. Results show that (a) awareness plays a central role in epidemic prevention; (b) a reasonable fraction of over-reacting nodes are needed in epidemic prevention; (c) R0 has different effects on epidemic outbreak for cases with and without asymptomatic infection; (d) social influence on behavior change can remarkably decrease the epidemic outbreak size. This research indicates that the media and opinion leaders should not understate the transmissibility and severity of diseases to ensure that people could become aware of the disease and adopt self-protection to protect themselves and the whole population.
Although there is always an interplay between the dynamics of information diffusion and disease spreading, the empirical research on the systemic coevolution mechanisms connecting these two spreading dynamics is still lacking. Here we investigate the coevolution mechanisms and dynamics between information and disease spreading by utilizing real data and a proposed spreading model on multiplex network. Our empirical analysis finds asymmetrical interactions between the information and disease spreading dynamics. Our results obtained from both the theoretical framework and extensive stochastic numerical simulations suggest that an information outbreak can be triggered in a communication network by its own spreading dynamics or by a disease outbreak on a contact network, but that the disease threshold is not affected by information spreading. Our key finding is that there is an optimal information transmission rate that markedly suppresses the disease spreading. We find that the time evolution of the dynamics in the proposed model qualitatively agrees with the real-world spreading processes at the optimal information transmission rate.
This paper mainly discusses the diffusion on complex networks with time-varying couplings. We propose a model to describe the adaptive diffusion process of local topological and dynamical information, and find that the Barabasi-Albert scale-free network (BA network) is beneficial to the diffusion and leads nodes to arrive at a larger state value than other networks do. The ability of diffusion for a node is related to its own degree. Specifically, nodes with smaller degrees are more likely to change their states and reach larger values, while those with larger degrees tend to stick to their original states. We introduce state entropy to analyze the thermodynamic mechanism of the diffusion process, and interestingly find that this kind of diffusion process is a minimization process of state entropy. We use the inequality constrained optimization method to reveal the restriction function of the minimization and find that it has the same form as the Gibbs free energy. The thermodynamical concept allows us to understand dynamical processes on complex networks from a brand-new perspective. The result provides a convenient means of optimizing relevant dynamical processes on practical circuits as well as related complex systems.
The impact that information diffusion has on epidemic spreading has recently attracted much attention. As a disease begins to spread in the population, information about the disease is transmitted to others, which in turn has an effect on the spread of disease. In this paper, using empirical results of the propagation of H7N9 and information about the disease, we clearly show that the spreading dynamics of the two-types of processes influence each other. We build a mathematical model in which both types of spreading dynamics are described using the SIS process in order to illustrate the influence of information diffusion on epidemic spreading. Both the simulation results and the pairwise analysis reveal that information diffusion can increase the threshold of an epidemic outbreak, decrease the final fraction of infected individuals and significantly decrease the rate at which the epidemic propagates. Additionally, we find that the multi-outbreak phenomena of epidemic spreading, along with the impact of information diffusion, is consistent with the empirical results. These findings highlight the requirement to maintain social awareness of diseases even when the epidemics seem to be under control in order to prevent a subsequent outbreak. These results may shed light on the in-depth understanding of the interplay between the dynamics of epidemic spreading and information diffusion.
Recently, information transmission models motivated by the classical epidemic propagation, have been applied to a wide-range of social systems, generally assume that information mainly transmits among individuals via peer-to-peer interactions on social networks. In this paper, we consider one more approach for users to get information: the out-of-social-network influence. Empirical analyses of eight typical events diffusion on a very large micro-blogging system, emph{Sina Weibo}, show that the external influence has significant impact on information spreading along with social activities. In addition, we propose a theoretical model to interpret the spreading process via both internal and external channels, considering three essential properties: (i) memory effect; (ii) role of spreaders; and (iii) non-redundancy of contacts. Experimental and mathematical results indicate that the information indeed spreads much quicker and broader with mutual effects of the internal and external influences. More importantly, the present model reveals that the event characteristic would highly determine the essential spreading patterns once the network structure is established. The results may shed some light on the in-depth understanding of the underlying dynamics of information transmission on real social networks.
We investigate the impact of community structure on information diffusion with the linear threshold model. Our results demonstrate that modular structure may have counter-intuitive effects on information diffusion when social reinforcement is present. We show that strong communities can facilitate global diffusion by enhancing local, intra-community spreading. Using both analytic approaches and numerical simulations, we demonstrate the existence of an optimal network modularity, where global diffusion require the minimal number of early adopters.