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Register a new userInfection percolation: A dynamic network model of disease spreading
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Models of disease spreading are critical for predicting infection growth in a population and evaluating public health policies. However, standard models typically represent the dynamics of disease transmission between individuals using macroscopic parameters that do not accurately represent person-to-person variability. To address this issue, we present a dynamic network model that provides a straightforward way to incorporate both disease transmission dynamics at the individual scale as well as the full spatiotemporal history of infection at the population scale. We find that disease spreads through a social network as a traveling wave of infection, followed by a traveling wave of recovery, with the onset and dynamics of spreading determined by the interplay between disease transmission and recovery. We use these insights to develop a scaling theory that predicts the dynamics of infection for diverse diseases and populations. Furthermore, we show how spatial heterogeneities in susceptibility to infection can either exacerbate or quell the spread of disease, depending on its infectivity. Ultimately, our dynamic network approach provides a simple way to model disease spreading that unifies previous findings and can be generalized to diverse diseases, containment strategies, seasonal conditions, and community structures.
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In this work we study a modified Susceptible-Infected-Susceptible (SIS) model in which the infection rate $lambda$ decays exponentially with the number of reinfections $n$, saturating after $n=l$. We find a critical decaying rate $epsilon_{c}(l)$ above which a finite fraction of the population becomes permanently infected. From the mean-field solution and computer simulations on hypercubic lattices we find evidences that the upper critical dimension is 6 like in the SIR model, which can be mapped in ordinary percolation.
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We analyze the dynamics of a population of independent random walkers on a graph and develop a simple model of epidemic spreading. We assume that each walker visits independently the nodes of a finite ergodic graph in a discrete-time markovian walk governed by his specific transition matrix. With this assumption, we first derive an upper bound for the reproduction numbers. Then we assume that a walker is in one of the states: susceptible, infectious, or recovered. An infectious walker remains infectious during a certain characteristic time. If an infectious walker meets a susceptible one on the same node there is a certain probability for the susceptible walker to get infected. By implementing this hypothesis in computer simulations we study the space-time evolution of the emerging infection patterns. Generally, random walk approaches seem to have a large potential to study epidemic spreading and to identify the pertinent parameters in epidemic dynamics.
We study a general epidemic model with arbitrary recovery rate distributions. This simple deviation from the standard setup is sufficient to prove that heterogeneity in the dynamical parameters can be as important as the more studied structural heterogeneity. Our analytical solution is able to predict the shift in the critical properties induced by heterogeneous recovery rates. Additionally, we show that the critical value of infectivity tends to be smaller than the one predicted by quenched mean-field approaches in the homogeneous case and that it can be linked to the variance of the recovery rates. We then illustrate the role of dynamical--structural correlations, which allow for a complete change in the critical behavior. We show that it is possible for a power-law network topology to behave similarly to a homogeneous structure by an appropriate tuning of its recovery rates, and vice versa. Finally, we show how heterogeneity in recovery rates affects the network localization properties of the spreading process.
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