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Unconditional Quantile Regression with High Dimensional Data

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 Added by Takuya Ura
 Publication date 2020
  fields Economy
and research's language is English




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Credible counterfactual analysis requires high-dimensional controls. This paper considers estimation and inference for heterogeneous counterfactual effects with high-dimensional data. We propose a novel doubly robust score for double/debiased estimation and inference for the unconditional quantile regression (Firpo, Fortin, and Lemieux, 2009) as a measure of heterogeneous counterfactual marginal effects. We propose a multiplier bootstrap inference for the Lasso double/debiased estimator, and develop asymptotic theories to guarantee that the bootstrap works. Simulation studies support our theories. Applying the proposed method to Job Corps survey data, we find that i) marginal effects of counterfactually extending the duration of the exposure to the Job Corps program are globally positive across quantiles regardless of definitions of the treatment and outcome variables, and that ii) these counterfactual effects are larger for higher potential earners than lower potential earners regardless of whether we define the outcome as the level or its logarithm.



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Quantile regression has become a valuable tool to analyze heterogeneous covaraite-response associations that are often encountered in practice. The development of quantile regression methodology for high-dimensional covariates primarily focuses on examination of model sparsity at a single or multiple quantile levels, which are typically pre-specified ad hoc by the users. The resulting models may be sensitive to the specific choices of the quantile levels, leading to difficulties in interpretation and erosion of confidence in the results. In this article, we propose a new penalization framework for quantile regression in the high-dimensional setting. We employ adaptive L1 penalties, and more importantly, propose a uniform selector of the tuning parameter for a set of quantile levels to avoid some of the potential problems with model selection at individual quantile levels. Our proposed approach achieves consistent shrinkage of regression quantile estimates across a continuous range of quantiles levels, enhancing the flexibility and robustness of the existing penalized quantile regression methods. Our theoretical results include the oracle rate of uniform convergence and weak convergence of the parameter estimators. We also use numerical studies to confirm our theoretical findings and illustrate the practical utility of our proposal
The widespread use of quantile regression methods depends crucially on the existence of fast algorithms. Despite numerous algorithmic improvements, the computation time is still non-negligible because researchers often estimate many quantile regressions and use the bootstrap for inference. We suggest two new fast algorithms for the estimation of a sequence of quantile regressions at many quantile indexes. The first algorithm applies the preprocessing idea of Portnoy and Koenker (1997) but exploits a previously estimated quantile regression to guess the sign of the residuals. This step allows for a reduction of the effective sample size. The second algorithm starts from a previously estimated quantile regression at a similar quantile index and updates it using a single Newton-Raphson iteration. The first algorithm is exact, while the second is only asymptotically equivalent to the traditional quantile regression estimator. We also apply the preprocessing idea to the bootstrap by using the sample estimates to guess the sign of the residuals in the bootstrap sample. Simulations show that our new algorithms provide very large improvements in computation time without significant (if any) cost in the quality of the estimates. For instance, we divide by 100 the time required to estimate 99 quantile regressions with 20 regressors and 50,000 observations.
This paper provides a method to construct simultaneous confidence bands for quantile functions and quantile effects in nonlinear network and panel models with unobserved two-way effects, strictly exogenous covariates, and possibly discrete outcome variables. The method is based upon projection of simultaneous confidence bands for distribution functions constructed from fixed effects distribution regression estimators. These fixed effects estimators are debiased to deal with the incidental parameter problem. Under asymptotic sequences where both dimensions of the data set grow at the same rate, the confidence bands for the quantile functions and effects have correct joint coverage in large samples. An empirical application to gravity models of trade illustrates the applicability of the methods to network data.
With the availability of high dimensional genetic biomarkers, it is of interest to identify heterogeneous effects of these predictors on patients survival, along with proper statistical inference. Censored quantile regression has emerged as a powerful tool for detecting heterogeneous effects of covariates on survival outcomes. To our knowledge, there is little work available to draw inference on the effects of high dimensional predictors for censored quantile regression. This paper proposes a novel procedure to draw inference on all predictors within the framework of global censored quantile regression, which investigates covariate-response associations over an interval of quantile levels, instead of a few discrete values. The proposed estimator combines a sequence of low dimensional model estimates that are based on multi-sample splittings and variable selection. We show that, under some regularity conditions, the estimator is consistent and asymptotically follows a Gaussian process indexed by the quantile level. Simulation studies indicate that our procedure can properly quantify the uncertainty of the estimates in high dimensional settings. We apply our method to analyze the heterogeneous effects of SNPs residing in lung cancer pathways on patients survival, using the Boston Lung Cancer Survival Cohort, a cancer epidemiology study on the molecular mechanism of lung cancer.
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