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The size distributions of power outages are shown to depend on the stress, or the proximity of the load of an electrical grid to complete breakdown. Using the data for the U.S. between 2002-2017, we show that the outage statistics are dependent on the usage levels during different hours of the day and months of the year. At higher load, not only are more failures likely, but the distribution of failure sizes shifts, to favor larger events. At a finer spatial scale, different regions within the U.S. can be shown to respond differently in terms of the outage statistics to variations in the usage (load). The response, in turn, corresponds to the respective bias towards larger or smaller failures in those regions. We provide a simple model, using realistic grid topologies, which can nonetheless demonstrate biases as a function of the applied load, as in the data. Given sufficient data of small scale events, the method can be used to identify vulnerable regions in power grids prior to major blackouts.
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Electric power-systems are one of the most important critical infrastructures. In recent years, they have been exposed to extreme stress due to the increasing demand, the introduction of distributed renewable energy sources, and the development of extensive interconnections. We investigate the phenomenon of abrupt breakdown of an electric power-system under two scenarios: load growth (mimicking the ever-increasing customer demand) and power fluctuations (mimicking the effects of renewable sources). Our results indicate that increasing the system size causes breakdowns to become more abrupt; in fact, mapping the system to a solvable statistical-physics model indicates the occurrence of a first order transition in the large size limit. Such an enhancement for the systemic risk failures (black-outs) with increasing network size is an effect that should be considered in the current projects aiming to integrate national power-grids into super-grids.
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For a power system operating in the vicinity of the power transfer limit of its transmission system, effect of stochastic fluctuations of power loads can become critical as a sufficiently strong such fluctuation may activate voltage instability and lead to a large scale collapse of the system. Considering the effect of these stochastic fluctuations near a codimension 1 saddle-node bifurcation, we explicitly calculate the autocorrelation function of the state vector and show how its behavior explains the phenomenon of critical slowing-down often observed for power systems on the threshold of blackout. We also estimate the collapse probability/mean clearing time for the power system and construct a new indicator function signaling the proximity to a large scale collapse. The new indicator function is easy to estimate in real time using PMU data feeds as well as SCADA information about fluctuations of power load on the nodes of the power grid. We discuss control strategies leading to the minimization of the collapse probability.
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Mathematical models are formal and simplified representations of the knowledge related to a phenomenon. In classical epidemic models, a neglected aspect is the heterogeneity of disease transmission and progression linked to the viral load of each infectious individual. Here, we attempt to investigate the interplay between the evolution of individuals viral load and the epidemic dynamics from a theoretical point of view. In the framework of multi-agent systems, we propose a particle stochastic model describing the infection transmission through interactions among agents and the individual physiological course of the disease. Agents have a double microscopic state: a discrete label, that denotes the epidemiological compartment to which they belong and switches in consequence of a Markovian process, and a microscopic trait, representing a normalized measure of their viral load, that changes in consequence of binary interactions or interactions with a background. Specifically, we consider Susceptible--Infected--Removed--like dynamics where infectious individuals may be isolated from the general population and the isolation rate may depend on the viral load sensitivity and frequency of tests. We derive kinetic evolution equations for the distribution functions of the viral load of the individuals in each compartment, whence, via suitable upscaling procedures, we obtain a macroscopic model for the densities and viral load momentum. We perform then a qualitative analysis of the ensuing macroscopic model, and we present numerical tests in the case of both constant and viral load-dependent isolation control. Also, the matching between the aggregate trends obtained from the macroscopic descriptions and the original particle dynamics simulated by a Monte Carlo approach is investigated.
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