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Register a new userEpidemic Dynamics On Information-Driven Adaptive Networks
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can evolve simultaneously. For the information-driven adaptive process, susceptible (infected) individuals who have abilities to recognize the disease would break the links of their infected (susceptible) neighbors to prevent the epidemic from further spreading. Simulation results and numerical analyses based on the pairwise approach indicate that the information-driven adaptive process can not only slow down the speed of epidemic spreading, but can also diminish the epidemic prevalence at the final state significantly. In addition, the disease spreading and information diffusion pattern on the lattice give a visual representation about how the disease is trapped into an isolated field with the information-driven adaptive process. Furthermore, we perform the local bifurcation analysis on four types of dynamical regions, including healthy, oscillatory, bistable and endemic, to understand the evolution of the observed dynamical behaviors. This work may shed some lights on understanding how information affects human activities on responding to epidemic spreading.
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There is currently growing interest in modeling the information diffusion on social networks across multi-disciplines. The majority of the corresponding research has focused on information diffusion independently, ignoring the network evolution in the diffusion process. Therefore, it is more reasonable to describe the real diffusion systems by the co-evolution between network topologies and information states. In this work, we propose a mechanism considering the coevolution between information states and network topology simultaneously, in which the information diffusion was executed as an SIS process and network topology evolved based on the adaptive assumption. The theoretical analyses based on the Markov approach were very consistent with simulation. Both simulation results and theoretical analyses indicated that the adaptive process, in which informed individuals would rewire the links between the informed neighbors to a random non-neighbor node, can enhance information diffusion (leading to much broader spreading). In addition, we obtained that two threshold values exist for the information diffusion on adaptive networks, i.e., if the information propagation probability is less than the first threshold, information cannot diffuse and dies out immediately; if the propagation probability is between the first and second threshold, information will spread to a finite range and die out gradually; and if the propagation probability is larger than the second threshold, information will diffuse to a certain size of population in the network. These results may shed some light on understanding the co-evolution between information diffusion and network topology.
We consider an epidemic process on adaptive activity-driven temporal networks, with adaptive behaviour modelled as a change in activity and attractiveness due to infection. By using a mean-field approach, we derive an analytical estimate of the epidemic threshold for SIS and SIR epidemic models for a general adaptive strategy, which strongly depends on the correlations between activity and attractiveness in the susceptible and infected states. We focus on strong social distancing, implementing two types of quarantine inspired by recent real case studies: an active quarantine, in which the population compensates the loss of links rewiring the ineffective connections towards non-quarantining nodes, and an inactive quarantine, in which the links with quarantined nodes are not rewired. Both strategies feature the same epidemic threshold but they strongly differ in the dynamics of active phase. We show that the active quarantine is extremely less effective in reducing the impact of the epidemic in the active phase compared to the inactive one, and that in SIR model a late adoption of measures requires inactive quarantine to reach containment.
Identifying super-spreaders in epidemics is important to suppress the spreading of disease especially when the medical resource is limited.In the modern society, the information on epidemics transmits swiftly through various communication channels which contributes much to the suppression of epidemics. Here we study on the identification of super-spreaders in the information-disease coupled spreading dynamics. Firstly, we find that the centralities in physical contact layer are no longer effective to identify super-spreaders in epidemics, which is due to the suppression effects from the information spreading. Then by considering the structural and dynamical couplings between the communication layer and physical contact layer, we propose a centrality measure called coupling-sensitive centrality to identify super-spreaders in disease spreading. Simulation results on synthesized and real-world multiplex networks show that the proposed measure is not only much more accurate than centralities on the single network, but also outperforms two typical multilayer centralities in identifying super-spreaders. These findings imply that considering the structural and dynamical couplings between layers is very necessary in identifying the key roles in the coupled multilayer systems.
Vaccination is an important measure available for preventing or reducing the spread of infectious diseases. In this paper, an epidemic model including susceptible, infected, and imperfectly vaccinated compartments is studied on Watts-Strogatz small-world, Barabasi-Albert scale-free, and random scale-free networks. The epidemic threshold and prevalence are analyzed. For small-world networks, the effective vaccination intervention is suggested and its influence on the threshold and prevalence is analyzed. For scale-free networks, the threshold is found to be strongly dependent both on the effective vaccination rate and on the connectivity distribution. Moreover, so long as vaccination is effective, it can linearly decrease the epidemic prevalence in small-world networks, whereas for scale-free networks it acts exponentially. These results can help in adopting pragmatic treatment upon diseases in structured populations.
We study SIS epidemic spreading processes unfolding on a recent generalisation of the activity-driven modelling framework. In this model of time-varying networks each node is described by two variables: activity and attractiveness. The first, describes the propensity to form connections. The second, defines the propensity to attract them. We derive analytically the epidemic threshold considering the timescale driving the evolution of contacts and the contagion as comparable. The solutions are general and hold for any joint distribution of activity and attractiveness. The theoretical picture is confirmed via large-scale numerical simulations performed considering heterogeneous distributions and different correlations between the two variables. We find that heterogeneous distributions of attractiveness alter the contagion process. In particular, in case of uncorrelated and positive correlations between the two variables, heterogeneous attractiveness facilitates the spreading. On the contrary, negative correlations between activity and attractiveness hamper the spreading. The results presented contribute to the understanding of the dynamical properties of time-varying networks and their effects on contagion phenomena unfolding on their fabric.
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