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Multilayer networks represent systems in which there are several topological levels each one representing one kind of interaction or interdependency between the systems elements. These networks have attracted a lot of attention recently because their study allows considering different dynamical modes concurrently. Here, we revise the main concepts and tools developed up to date. Specifically, we focus on several metrics for multilayer network characterization as well as on the spectral properties of the system, which ultimately enable for the dynamical characterization of several critical phenomena. The theoretical framework is also applied for description of real-world multilayer systems.
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The controllability of a network is a theoretical problem of relevance in a variety of contexts ranging from financial markets to the brain. Until now, network controllability has been characterized only on isolated networks, while the vast majority of complex systems are formed by multilayer networks. Here we build a theoretical framework for the linear controllability of multilayer networks by mapping the problem into a combinatorial matching problem. We found that correlating the external signals in the different layers can significantly reduce the multiplex network robustness to node removal, as it can be seen in conjunction with a hybrid phase transition occurring in interacting Poisson networks. Moreover we observe that multilayer networks can stabilize the fully controllable multiplex network configuration that can be stable also when the full controllability of the single network is not stable.
Spreading processes have been largely studied in the literature, both analytically and by means of large-scale numerical simulations. These processes mainly include the propagation of diseases, rumors and information on top of a given population. In the last two decades, with the advent of modern network science, we have witnessed significant advances in this field of research. Here we review the main theoretical and numerical methods developed for the study of spreading processes on complex networked systems. Specifically, we formally define epidemic processes on single and multilayer networks and discuss in detail the main methods used to perform numerical simulations. Throughout the review, we classify spreading processes (disease and rumor models) into two classes according to the nature of time: (i) continuous-time and (ii) cellular automata approach, where the second one can be further divided into synchronous and asynchronous updating schemes. Our revision includes the heterogeneous mean-field, the quenched-mean field, and the pair quenched mean field approaches, as well as their respective simulation techniques, emphasizing similarities and differences among the different techniques. The content presented here offers a whole suite of methods to study epidemic-like processes in complex networks, both for researchers without previous experience in the subject and for experts.
The largest eigenvalue of a networks adjacency matrix and its associated principal eigenvector are key elements for determining the topological structure and the properties of dynamical processes mediated by it. We present a physically grounded expression relating the value of the largest eigenvalue of a given network to the largest eigenvalue of two network subgraphs, considered as isolated: The hub with its immediate neighbors and the densely connected set of nodes with maximum $K$-core index. We validate this formula showing that it predicts with good accuracy the largest eigenvalue of a large set of synthetic and real-world topologies. We also present evidence of the consequences of these findings for broad classes of dynamics taking place on the networks. As a byproduct, we reveal that the spectral properties of heterogeneous networks built according to the linear preferential attachment model are qualitatively different from those of their static counterparts.
We propose a network metric, edge proximity, ${cal P}_e$, which demonstrates the importance of specific edges in a network, hitherto not captured by existing network metrics. The effects of removing edges with high ${cal P}_e$ might initially seem inconspicuous but are eventually shown to be very harmful for networks. Compared to existing strategies, the removal of edges by ${cal P}_e$ leads to a remarkable increase in the diameter and average shortest path length in undirected real and random networks till the first disconnection and well beyond. ${cal P}_e$ can be consistently used to rupture the network into two nearly equal parts, thus presenting a very potent strategy to greatly harm a network. Targeting by ${cal P}_e$ causes notable efficiency loss in U.S. and European power grid networks. ${cal P}_e$ identifies proteins with essential cellular functions in protein-protein interaction networks. It pinpoints regulatory neural connections and important portions of the neural and brain networks, respectively. Energy flow interactions identified by ${cal P}_e$ form the backbone of long food web chains. Finally, we scrutinize the potential of ${cal P}_e$ in edge controllability dynamics of directed networks.
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