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Register a new userStochastic dynamical model of a growing network based on self-exciting point process
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We perform experimental verification of the preferential attachment model that is commonly accepted as a generating mechanism of the scale-free complex networks. To this end we chose citation network of Physics papers and traced citation history of 40,195 papers published in one year. Contrary to common belief, we found that citation dynamics of the individual papers follows the emph{superlinear} preferential attachment, with the exponent $alpha= 1.25-1.3$. Moreover, we showed that the citation process cannot be described as a memoryless Markov chain since there is substantial correlation between the present and recent citation rates of a paper. Basing on our findings we constructed a stochastic growth model of the citation network, performed numerical simulations based on this model and achieved an excellent agreement with the measured citation distributions.
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In citation networks, the activity of papers usually decreases with age and dormant papers may be discovered and become fashionable again. To model this phenomenon, a competition mechanism is suggested which incorporates two factors: vigorousness and dormancy. Based on this idea, a citation network model is proposed, in which a node has two discrete stage: vigorous and dormant. Vigorous nodes can be deactivated and dormant nodes may be activated and become vigorous. The evolution of the network couples addition of new nodes and state transitions of old ones. Both analytical calculation and numerical simulation show that the degree distribution of nodes in generated networks displays a good right-skewed behavior. Particularly, scale-free networks are obtained as the deactivated vertex is target selected and exponential networks are realized for the random-selected case. Moreover, the measurement of four real-world citation networks achieves a good agreement with the stochastic model.
We present a detailed analysis of the self-organization phenomenon in which the stylized facts originate from finite size effects with respect to the number of agents considered and disappear in the limit of an infinite population. By introducing the possibility that agents can enter or leave the market depending on the behavior of the price, it is possible to show that the system self-organizes in a regime with a finite number of agents which corresponds to the stylized facts. The mechanism to enter or leave the market is based on the idea that a too stable market is unappealing for traders while the presence of price movements attracts agents to enter and speculate on the market. We show that this mechanism is also compatible with the idea that agents are scared by a noisy and risky market at shorter time scales. We also show that the mechanism for self-organization is robust with respect to variations of the exit/entry rules and that the attempt to trigger the system to self-organize in a region without stylized facts leads to an unrealistic dynamics. We study the self-organization in a specific agent based model but we believe that the basic ideas should be of general validity.
Models of disease spreading are critical for predicting infection growth in a population and evaluating public health policies. However, standard models typically represent the dynamics of disease transmission between individuals using macroscopic parameters that do not accurately represent person-to-person variability. To address this issue, we present a dynamic network model that provides a straightforward way to incorporate both disease transmission dynamics at the individual scale as well as the full spatiotemporal history of infection at the population scale. We find that disease spreads through a social network as a traveling wave of infection, followed by a traveling wave of recovery, with the onset and dynamics of spreading determined by the interplay between disease transmission and recovery. We use these insights to develop a scaling theory that predicts the dynamics of infection for diverse diseases and populations. Furthermore, we show how spatial heterogeneities in susceptibility to infection can either exacerbate or quell the spread of disease, depending on its infectivity. Ultimately, our dynamic network approach provides a simple way to model disease spreading that unifies previous findings and can be generalized to diverse diseases, containment strategies, seasonal conditions, and community structures.
In this paper, we propose a Boltzmann-type kinetic model of the spreading of an infectious disease on a network. The latter describes the connections among countries, cities or districts depending on the spatial scale of interest. The disease transmission is represented in terms of the viral load of the individuals and is mediated by social contacts among them, taking into account their displacements across the nodes of the network. We formally derive the hydrodynamic equations for the density and the mean viral load of the individuals on the network and we analyse the large-time trends of these quantities with special emphasis on the cases of blow-up or eradication of the infection. By means of numerical tests, we also investigate the impact of confinement measures, such as quarantine or localised lockdown, on the diffusion of the disease on the network.
Here we provide a detailed analysis, along with some extensions and additonal investigations, of a recently proposed self-organised model for the evolution of complex networks. Vertices of the network are characterised by a fitness variable evolving through an extremal dynamics process, as in the Bak-Sneppen model representing a prototype of Self-Organized Criticality. The network topology is in turn shaped by the fitness variable itself, as in the fitness network model. The system self-organizes to a nontrivial state, characterized by a power-law decay of dynamical and topological quantities above a critical threshold. The interplay between topology and dynamics in the system is the key ingredient leading to an unexpected behaviour of these quantities.
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