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Register a new userThe Aging Effect in Evolving Scientific Citation Networks
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The study of citation networks is of interest to the scientific community. However, the underlying mechanism driving individual citation behavior remains imperfectly understood, despite the recent proliferation of quantitative research methods. Traditional network models normally use graph theory to consider articles as nodes and citations as pairwise relationships between them. In this paper, we propose an alternative evolutionary model based on hypergraph theory in which one hyperedge can have an arbitrary number of nodes, combined with an aging effect to reflect the temporal dynamics of scientific citation behavior. Both theoretical approximate solution and simulation analysis of the model are developed and validated using two benchmark datasets from different disciplines, i.e. publications of the American Physical Society (APS) and the Digital Bibliography & Library Project (DBLP). Further analysis indicates that the attraction of early publications will decay exponentially. Moreover, the experimental results show that the aging effect indeed has a significant influence on the description of collective citation patterns. Shedding light on the complex dynamics driving these mechanisms facilitates the understanding of the laws governing scientific evolution and the quantitative evaluation of scientific outputs.
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Structural inequalities persist in society, conferring systematic advantages to some people at the expense of others, for example, by giving them substantially more influence and opportunities. Using bibliometric data about authors of scientific publications, we identify two types of structural inequalities in scientific citations. First, female authors, who represent a minority of researchers, receive less recognition for their work (through citations) relative to male authors; second, authors affiliated with top-ranked institutions, who are also a minority, receive substantially more recognition compared to other authors. We present a model for the growth of directed citation networks and show that citations disparities arise from individual preferences to cite authors from the same group (homophily), highly cited or active authors (preferential attachment), as well as the size of the group and how frequently new authors join. We analyze the model and show that its predictions align well with real-world observations. Our theoretical and empirical analysis also suggests potential strategies to mitigate structural inequalities in science. In particular, we find that merely increasing the minority group size does little to narrow the disparities. Instead, reducing the homophily of each group, frequently adding new authors to a research field while providing them an accessible platform among existing, established authors, together with balanced group sizes can have the largest impact on reducing inequality. Our work highlights additional complexities of mitigating structural disparities stemming from asymmetric relations (e.g., directed citations) compared to symmetric relations (e.g., collaborations).
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Time-varying network topologies can deeply influence dynamical processes mediated by them. Memory effects in the pattern of interactions among individuals are also known to affect how diffusive and spreading phenomena take place. In this paper we analyze the combined effect of these two ingredients on epidemic dynamics on networks. We study the susceptible-infected-susceptible (SIS) and the susceptible-infected-removed (SIR) models on the recently introduced activity-driven networks with memory. By means of an activity-based mean-field approach we derive, in the long time limit, analytical predictions for the epidemic threshold as a function of the parameters describing the distribution of activities and the strength of the memory effects. Our results show that memory reduces the threshold, which is the same for SIS and SIR dynamics, therefore favouring epidemic spreading. The theoretical approach perfectly agrees with numerical simulations in the long time asymptotic regime. Strong aging effects are present in the preasymptotic regime and the epidemic threshold is deeply affected by the starting time of the epidemics. We discuss in detail the origin of the model-dependent preasymptotic corrections, whose understanding could potentially allow for epidemic control on correlated temporal networks.
Motivated by results of Henry, Pralat and Zhang (PNAS 108.21 (2011): 8605-8610), we propose a general scheme for evolving spatial networks in order to reduce their total edge lengths. We study the properties of the equilbria of two networks from this class, which interpolate between three well studied objects: the ErdH{o}s-R{e}nyi random graph, the random geometric graph, and the minimum spanning tree. The first of our two evolutions can be used as a model for a social network where individuals have fixed opinions about a number of issues and adjust their ties to be connected to people with similar views. The second evolution which preserves the connectivity of the network has potential applications in the design of transportation networks and other distribution systems.
Modularity structures are common in various social and biological networks. However, its dynamical origin remains an open question. In this work, we set up a dynamical model describing the evolution of a social network. Based on the observations of real social networks, we introduced a link-creating/deleting strategy according to the local dynamics in the model. Thus the coevolution of dynamics and topology naturally determines the network properties. It is found that for a small coupling strength, the networked system cannot reach any synchronization and the network topology is homogeneous. Interestingly, when the coupling strength is large enough, the networked system spontaneously forms communities with different dynamical states. Meanwhile, the network topology becomes heterogeneous with modular structures. It is further shown that in a certain parameter regime, both the degree and the community size in the formed network follow a power-law distribution, and the networks are found to be assortative. These results are consistent with the characteristics of many empirical networks, and are helpful to understand the mechanism of formation of modularity in complex networks.
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