No Arabic abstract
Network structure can affect when and how widely new ideas, products, and behaviors are adopted. In widely-used models of biological contagion, interventions that randomly rewire edges (generally making them longer) accelerate spread. However, there are other models relevant to social contagion, such as those motivated by myopic best-response in games with strategic complements, in which an individuals behavior is described by a threshold number of adopting neighbors above which adoption occurs (i.e., complex contagions). Recent work has argued that highly clustered, rather than random, networks facilitate spread of these complex contagions. Here we show that minor modifications to this model, which make it more realistic, reverse this result: we allow very rare below-threshold adoption, i.e., rarely adoption occurs when there is only one adopting neighbor. To model the trade-off between long and short edges we consider networks that are the union of cycle-power-$k$ graphs and random graphs on $n$ nodes. Allowing adoptions below threshold to occur with order $1/sqrt{n}$ probability along some short cycle edges is enough to ensure that random rewiring accelerates spread. Simulations illustrate the robustness of these results to other commonly-posited models for noisy best-response behavior. Hypothetical interventions that randomly rewire existing edges or add random edges (versus adding short, triad-closing edges) in hundreds of empirical social networks reduce time to spread. This revised conclusion suggests that those wanting to increase spread should induce formation of long ties, rather than triad-closing ties. More generally, this highlights the importance of noise in game-theoretic analyses of behavior.
Given a set of attributed subgraphs known to be from different classes, how can we discover their differences? There are many cases where collections of subgraphs may be contrasted against each other. For example, they may be assigned ground truth labels (spam/not-spam), or it may be desired to directly compare the biological networks of different species or compound networks of different chemicals. In this work we introduce the problem of characterizing the differences between attributed subgraphs that belong to different classes. We define this characterization problem as one of partitioning the attributes into as many groups as the number of classes, while maximizing the total attributed quality score of all the given subgraphs. We show that our attribute-to-class assignment problem is NP-hard and an optimal $(1 - 1/e)$-approximation algorithm exists. We also propose two different faster heuristics that are linear-time in the number of attributes and subgraphs. Unlike previous work where only attributes were taken into account for characterization, here we exploit both attributes and social ties (i.e. graph structure). Through extensive experiments, we compare our proposed algorithms, show findings that agree with human intuition on datasets from Amazon co-purchases, Congressional bill sponsorships, and DBLP co-authorships. We also show that our approach of characterizing subgraphs is better suited for sense-making than discriminating classification approaches.
Since the publication of Complex Contagions and the Weakness of Long Ties in 2007, complex contagions have been studied across an enormous variety of social domains. In reviewing this decade of research, we discuss recent advancements in applied studies of complex contagions, particularly in the domains of health, innovation diffusion, social media, and politics. We also discuss how these empirical studies have spurred complementary advancements in the theoretical modeling of contagions, which concern the effects of network topology on diffusion, as well as the effects of individual-level attributes and thresholds. In synthesizing these developments, we suggest three main directions for future research. The first concerns the study of how multiple contagions interact within the same network and across networks, in what may be called an ecology of contagions. The second concerns the study of how the structure of thresholds and their behavioral consequences can vary by individual and social context. The third area concerns the roles of diversity and homophily in the dynamics of complex contagion, including both diversity of demographic profiles among local peers, and the broader notion of structural diversity within a network. Throughout this discussion, we make an effort to highlight the theoretical and empirical opportunities that lie ahead.
The spread of ideas across a social network can be studied using complex contagion models, in which agents are activated by contact with multiple activated neighbors. The investigation of complex contagions can provide crucial insights into social influence and behavior-adoption cascades on networks. In this paper, we introduce a model of a multi-stage complex contagion on networks. Agents at different stages --- which could, for example, represent differing levels of support for a social movement or differing levels of commitment to a certain product or idea --- exert different amounts of influence on their neighbors. We demonstrate that the presence of even one additional stage introduces novel dynamical behavior, including interplay between multiple cascades, that cannot occur in single-stage contagion models. We find that cascades --- and hence collective action --- can be driven not only by high-stage influencers but also by low-stage influencers.
Physical contacts result in the spread of various phenomena such as viruses, gossips, ideas, packages and marketing pamphlets across a population. The spread depends on how people move and co-locate with each other, or their mobility patterns. How far such phenomena spread has significance for both policy making and personal decision making, e.g., studying the spread of COVID-19 under different intervention strategies such as wearing a mask. In practice, mobility patterns of an entire population is never available, and we usually have access to location data of a subset of individuals. In this paper, we formalize and study the problem of estimating the spread of a phenomena in a population, given that we only have access to sub-samples of location visits of some individuals in the population. We show that simple solutions such as estimating the spread in the sub-sample and scaling it to the population, or more sophisticated solutions that rely on modeling location visits of individuals do not perform well in practice, the former because it ignores contacts between unobserved individuals and sampled ones and the latter because it yields inaccurate modeling of co-locations. Instead, we directly model the co-locations between the individuals. We introduce PollSpreader and PollSusceptible, two novel approaches that model the co-locations between individuals using a contact network, and infer the properties of the contact network using the subsample to estimate the spread of the phenomena in the entire population. We show that our estimates provide an upper bound and a lower bound on the spread of the disease in expectation. Finally, using a large high-resolution real-world mobility dataset, we experimentally show that our estimates are accurate, while other methods that do not correctly account for co-locations between individuals result in wrong observations (e.g, premature herd-immunity).
We study the evolution of cooperation in populations where individuals play prisoners dilemma on a network. Every node of the network corresponds on an individual choosing whether to cooperate or defect in a repeated game. The players revise their actions by imitating those neighbors who have higher payoffs. We show that when the interactions take place on graphs with large girth, cooperation is more likely to emerge. On the flip side, in graphs with many cycles of length 3 and 4, defection spreads more rapidly. One of the key ideas of our analysis is that our dynamics can be seen as a perturbation of the voter model. We write the transition kernel of the corresponding Markov chain in terms of the pairwise correlations in the voter model. We analyze the pairwise correlation and show that in graphs with relatively large girth, cooperators cluster and help each other.