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Register a new userThe Role of the Propensity Score in Fixed Effect Models
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We develop a new approach for estimating average treatment effects in the observational studies with unobserved group-level heterogeneity. A common approach in such settings is to use linear fixed effect specifications estimated by least squares regression. Such methods severely limit the extent of the heterogeneity between groups by making the restrictive assumption that linearly adjusting for differences between groups in average covariate values addresses all concerns with cross-group comparisons. We start by making two observations. First we note that the fixed effect method in effect adjusts only for differences between groups by adjusting for the average of covariate values and average treatment. Second, we note that weighting by the inverse of the propensity score would remove biases for comparisons between treated and control units under the fixed effect set up. We then develop three generalizations of the fixed effect approach based on these two observations. First, we suggest more general, nonlinear, adjustments for the average covariate values. Second, we suggest robustifying the estimators by using propensity score weighting. Third, we motivate and develop implementations for adjustments that also adjust for group characteristics beyond the average covariate values.
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The lack of longitudinal studies of the relationship between the built environment and travel behavior has been widely discussed in the literature. This paper discusses how standard propensity score matching estimators can be extended to enable such studies by pairing observations across two dimensions: longitudinal and cross-sectional. Researchers mimic randomized controlled trials (RCTs) and match observations in both dimensions, to find synthetic control groups that are similar to the treatment group and to match subjects synthetically across before-treatment and after-treatment time periods. We call this a two-dimensional propensity score matching (2DPSM). This method demonstrates superior performance for estimating treatment effects based on Monte Carlo evidence. A near-term opportunity for such matching is identifying the impact of transportation infrastructure on travel behavior.
Selective inference (post-selection inference) is a methodology that has attracted much attention in recent years in the fields of statistics and machine learning. Naive inference based on data that are also used for model selection tends to show an overestimation, and so the selective inference conditions the event that the model was selected. In this paper, we develop selective inference in propensity score analysis with a semiparametric approach, which has become a standard tool in causal inference. Specifically, for the most basic causal inference model in which the causal effect can be written as a linear sum of confounding variables, we conduct Lasso-type variable selection by adding an $ell_1$ penalty term to the loss function that gives a semiparametric estimator. Confidence intervals are then given for the coefficients of the selected confounding variables, conditional on the event of variable selection, with asymptotic guarantees. An important property of this method is that it does not require modeling of nonparametric regression functions for the outcome variables, as is usually the case with semiparametric propensity score analysis.
This paper proposes a logistic undirected network formation model which allows for assortative matching on observed individual characteristics and the presence of edge-wise fixed effects. We model the coefficients of observed characteristics to have a latent community structure and the edge-wise fixed effects to be of low rank. We propose a multi-step estimation procedure involving nuclear norm regularization, sample splitting, iterative logistic regression and spectral clustering to detect the latent communities. We show that the latent communities can be exactly recovered when the expected degree of the network is of order log n or higher, where n is the number of nodes in the network. The finite sample performance of the new estimation and inference methods is illustrated through both simulated and real datasets.
In nonseparable triangular models with a binary endogenous treatment and a binary instrumental variable, Vuong and Xu (2017) show that the individual treatment effects (ITEs) are identifiable. Feng, Vuong and Xu (2019) show that a kernel density estimator that uses nonparametrically estimated ITEs as observations is uniformly consistent for the density of the ITE. In this paper, we establish the asymptotic normality of the density estimator of Feng, Vuong and Xu (2019) and show that despite their faster rate of convergence, ITEs estimation errors have a non-negligible effect on the asymptotic distribution of the density estimator. We propose asymptotically valid standard errors for the density of the ITE that account for estimated ITEs as well as bias correction. Furthermore, we develop uniform confidence bands for the density of the ITE using nonparametric or jackknife multiplier bootstrap critical values. Our uniform confidence bands have correct coverage probabilities asymptotically with polynomial error rates and can be used for inference on the shape of the ITEs distribution.
We analyze the sources of changes in the distribution of hourly wages in the United States using CPS data for the survey years 1976 to 2019. We account for the selection bias from the employment decision by modeling the distribution of annual hours of work and estimating a nonseparable model of wages which uses a control function to account for selection. This allows the inclusion of all individuals working positive hours and provides a fuller description of the wage distribution. We decompose changes in the distribution of wages into composition, structural and selection effects. Composition effects have increased wages at all quantiles but the patterns of change are generally determined by the structural effects. Evidence of changes in the selection effects only appear at the lower quantiles of the female wage distribution. These various components combine to produce a substantial increase in wage inequality.
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