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Register a new userContrasting Effects of Strong Ties on SIR and SIS Processes in Temporal Networks
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Most real networks are characterized by connectivity patterns that evolve in time following complex, non-Markovian, dynamics. Here we investigate the impact of this ubiquitous feature by studying the Susceptible-Infected-Recovered (SIR) and Susceptible-Infected-Susceptible (SIS) epidemic models on activity driven networks with and without memory (i.e., Markovian and non-Markovian). We show that while memory inhibits the spreading process in SIR models, where the epidemic threshold is moved to larger values, it plays the opposite effect in the case of the SIS, where the threshold is lowered. The heterogeneity in tie strengths, and the frequent repetition of connections that it entails, allows in fact less virulent SIS-like diseases to survive in tightly connected local clusters that serve as reservoir for the virus. We validate this picture by evaluating the threshold of both processes in a real temporal network. Our findings confirm the important role played by non-Markovian network dynamics on dynamical processes
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Multi-layered networks represent a major advance in the description of natural complex systems, and their study has shed light on new physical phenomena. Despite its importance, however, the role of the temporal dimension in their structure and function has not been investigated in much detail so far. Here we study the temporal correlations between layers exhibited by real social multiplex networks. At a basic level, the presence of such correlations implies a certain degree of predictability in the contact pattern, as we quantify by an extension of the entropy and mutual information analyses proposed for the single-layer case. At a different level, we demonstrate that temporal correlations are a signature of a multitasking behavior of network agents, characterized by a higher level of switching between different social activities than expected in a uncorrelated pattern. Moreover, temporal correlations significantly affect the dynamics of coupled epidemic processes unfolding on the network. Our work opens the way for the systematic study of temporal multiplex networks and we anticipate it will be of interest to researchers in a broad array of fields.
Algorithms for search of communities in networks usually consist discrete variations of links. Here we discuss a flow method, driven by a set of differential equations. Two examples are demonstrated in detail. First is a partition of a signed graph into two parts, where the proposed equations are interpreted in terms of removal of a cognitive dissonance by agents placed in the network nodes. There, the signs and values of links refer to positive or negative interpersonal relationships of different strength. Second is an application of a method akin to the previous one, dedicated to communities identification, to the Sierpinski triangle of finite size. During the time evolution, the related graphs are weighted; yet at the end the discrete character of links is restored. In the case of the Sierpinski triangle, the method is supplemented by adding a small noise to the initial connectivity matrix. By breaking the symmetry of the network, this allows to a successful handling of overlapping nodes.
Weak ties play a significant role in the structures and the dynamics of community networks. Based on the susceptible-infected model in contact process, we study numerically how weak ties influence the predictability of epidemic dynamics. We first investigate the effects of different kinds of weak ties on the variabilities of both the arrival time and the prevalence of disease, and find that the bridgeness with small degree can enhance the predictability of epidemic spreading. Once weak ties are settled, compared with the variability of arrival time, the variability of prevalence displays a diametrically opposed changing trend with both the distance of the initial seed to the bridgeness and the degree of the initial seed. More specifically, the further distance and the larger degree of the initial seed can induce the better predictability of arrival time and the worse predictability of prevalence. Moreover, we discuss the effects of weak tie number on the epidemic variability. As community strength becomes very strong, which is caused by the decrease of weak tie number, the epidemic variability will change dramatically. Compared with the case of hub seed and random seed, the bridgenss seed can result in the worst predictability of arrival time and the best predictability of prevalence. These results show that the variability of arrival time always marks a complete reversal trend of that of prevalence, which implies it is impossible to predict epidemic spreading in the early stage of outbreaks accurately.
To improve the accuracy of network-based SIS models we introduce and study a multilayer representation of a time-dependent network. In particular, we assume that individuals have their long-term (permanent) contacts that are always present, identifying in this way the first network layer. A second network layer also exists, where the same set of nodes can be connected by occasional links, created with a given probability. While links of the first layer are permanent, a link of the second layer is only activated with some probability and under the condition that the two nodes, connected by this link, are simultaneously participating to the temporary link. We develop a model for the SIS epidemic on this time-dependent network, analyze equilibrium and stability of the corresponding mean-field equations, and shed some light on the role of the temporal layer on the spreading process.
Understanding spreading dynamics will benefit society as a whole in better preventing and controlling diseases, as well as facilitating the socially responsible information while depressing destructive rumors. In network-based spreading dynamics, edges with different weights may play far different roles: a friend from afar usually brings novel stories, and an intimate relationship is highly risky for a flu epidemic. In this article, we propose a weighted susceptible-infected-susceptible model on complex networks, where the weight of an edge is defined by the topological proximity of the two associated nodes. Each infected individual is allowed to select limited number of neighbors to contact, and a tunable parameter is introduced to control the preference to contact through high-weight or low-weight edges. Experimental results on six real networks show that the epidemic prevalence can be largely promoted when strong ties are favored in the spreading process. By comparing with two statistical null models respectively with randomized topology and randomly redistributed weights, we show that the distribution pattern of weights, rather than the topology, mainly contributes to the experimental observations. Further analysis suggests that the weight-weight correlation strongly affects the results: high-weight edges are more significant in keeping high epidemic prevalence when the weight-weight correlation is present.
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